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Q., Wang K
These studies often extend into computational modeling workflows, where experimental data are combined with structural predictions
While these medications effectively decrease acid levels, they fail to address the underlying mechanical dysfunction allowing reflux to occur
Calcium transients and BDNF secretion in response to activation of GPCRs and RTKs In addition to calcium influx through VGCC or NMDA receptors, calcium release from internal calcium stores is mediated by activation of G protein-coupled receptors (GPCRs) or receptor tyrosine kinases (RTKs)
