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Description
In SH-SY5Y cells, hMOR phosphorylation at S377 (the equivalent of S375 for the rMOR) upon DAMGO exposure does not rely on GRK2 suggesting the implication of another kinases (Mouldous et al., 2012)
The mechanisms of TB-500 are still being investigated, but known actions of TB4 include: Binding as an actin-binding protein to inhibit the polymerization of globular actin (G-actin) into filamentous actin (F-actin), known as actin sequestration, which results in higher G-actin levels and increased cellular motility [23, 24] TB4s ability to increase cellular motility may help progenitor cells to reach sites of injury and initiate tissue recovery [25]
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CHST14/D4ST1 deficiency: new form of EhlersDanlos syndrome
