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Description
This translates into mean increases of 1.99 and 1.36 min, respectively
L-type Ca 2+ channels (LTCCs) facilitate Ca 2+ influx during late myocyte depolarization, provoking the Ca 2+ -induced Ca 2+ release that sustains the action potential plateau and catalyzes myofilament interaction (Wu and Corr, 1992
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This mutation involves glycosaminoglycan metabolism and is characterized by severe multisystemic and rapidly progressive phenotype not treatable with enzymatic therapy
