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A recent study demonstrated that in mice somatostatin and glucagon secretion are linked in a reciprocal feedback cycle with somatostatin inhibiting glucagon secretion at low and high glucose levels, and glucagon stimulating somatostatin secretion via the glucagon and GLP-1 receptors

By targeting GLP-1 + GIP + Glucagon receptors, clinical studies have reported weight loss approaching 24% or more, making it one of the most effective obesity therapies under development

Compared to placebo, GLP-1RA agents generally showed numerically lower MACE risk in the established cardiovascular disease subgroup (including dulaglutide, efpeglenatide, exenatide, and liraglutide), with the exception of semaglutide