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5.3.3 Improvement of myocardial metabolism, associated protective effects, and indirect impact on CAD GLP-1RAs enhance mitochondrial function in cardiomyocytes, mitigate oxidative stress and cellular damage, improve myocardial fatty acid metabolism, stabilize blood glucose levels, and regulate myocardial mitochondrial function via the reninangiotensinaldosterone system (156, 157)

According to the FDA, there is no known lawful basis for using these salt forms in compounding, and the FDA is not aware of evidence that they have the same chemical and pharmacological properties as base-form semaglutide

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