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A better understanding of the physiopathogenic mechanisms involved in CR could lead to better treatment for these patients.3,4 OBJECTIVES In the present paper we reviewed the evidence pointing towards the cigarette smoke effects on the respiratory epithelium as well as its role on the pathophysiology of chronic rhinosinusitis

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Since the civet coffee bubble burst, the animals only tend make the news in discussions of potential reservoirs for transmissible-to-human diseases like SARS