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The other main driver mutations found in this subtype, RET fusion as well as mutations in NKX2-1, which regulates RET, were present in a small proportion and exclusive to piano tumors

Together, these observations suggest that the capacity to oxidize fatty acid and/or transport of fatty acyl-CoA by carnitine palmitoyltransferase I (CPT1) in the skeletal muscle was unaltered by chronic exposure to cigarette smoke and therefore do not support a significant role for the mitochondria in visceral fat accumulation with smoking (52)