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Description
This study extends our understanding of the molecular signaling circuits and cellular dynamics in smoking-induced lung inflammation and pathology, highlights the functional consequence of monocyte and neutrophil recruitment, identifies MoAM as key drivers of the inflammatory process, and supports their contribution to pathological tissue remodeling

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Elle ne reprsente pas une substance cancrigne comme lest, contrario, le monoxyde de carbone prsent dans les cigarettes traditionnelles