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Pathological phenotypes associated with biallelic variants of NADSYN lead to defects in multiple organs and limbs, and affected individuals do not usually survive for a few months after birth (Szot et al., 2020)
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They demonstrated that miR-365-3p/ETS homologous factor (EHF, a KRT16transcription factor) axis could reduce OSCC cell migration, invasion, metastasis, and chemoresistance to 5-fluorouracil through the suppression of KRT16
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