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The above considerations suggest that two pharmacologically distinct mechanisms with opposite effects on the same signaling pathway are involved in a normally unresolvable physiological failure (complete obstruction (ligation) of vessels and ischemia due to a cause that could be not removed, inadequate rescue and unable to heal)

Fliers, E., et al

The growth characteristics of the double disruption strain (P1- IspedEIspedH ) suggest that additional Pr 3+ -induced EG dehydrogenases, including Is XoxF, may also participate in this metabolic process

This differential regulation allows therapeutic benefits without common side effects associated with NO manipulation