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We have produced a large-scale computational biology analysis of the SNO-proteome in the cortex of Shank3 mutant mice (a popular model of ASD [171, 282, 283]) and found that among the NO-related molecular alterations, the most prominent change was the activation of the mTOR signaling pathway [171, 176]

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Conversely, we found a significant difference in HbA1c levels before and after the introduction of GLP-1 RAs (MD 0.79
