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LKB1 deletion promotes the expression of hypoxia-inducible factor (HIF)-1 and stabilizes it by regulating mTOR activity in NSCLC cells

The absence of human teratogenicity data means risk cannot be quantified, but early first-trimester exposure coincides with organogenesis when developmental signaling is most vulnerable to external modulators

This antioxidative potential arises from its ability to neutralize free radicals, particularly when regenerated by co-antioxidants like vitamin C and glutathione, which stabilize its effects and prevent the loss of its antioxidative capacity 177

(2009) Effects of Japanese herbal medicine, Kampo, on human UGT1A1 activity