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FMA Publications Short article series with inside updates, outcomes, and follow-ups related to the rebuild of FMA

Autophagy is a self-protective mechanism that maintains cellular homeostasis and promotes cell survival by blocking apoptosis pathways [200,201,202]

It is worth noting that nAbs, through their interaction with the bodys T cells, are able to eliminate cells transduced by AAV, causing inflammation in the target organ and reducing gene transfer efficiency and duration [106]

This aggregation is further enhanced by the addition of excess copper, which accelerates the overall aggregation of A 142 in comparison to A 140 (Jin et al., 2011)
