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Early wound healing studies demonstrated that GHK-Cu application to experimental wounds in animals accelerated healing, increased collagen deposition, enhanced wound breaking strength, improved tissue organization, and reduced scarring compared to controls

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Taken together, these findings suggest that APR-246 could induce tumor cell death through both reactivation of mutant p53 and inhibition of cellular thiol-dependent redox system proteins (GPX4, TXNDR1, GRX1), providing novel combinatorial strategies for AML therapy