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These findings suggest that adequate exportation of Gln from astrocytes represents a crucial regulatory point for maintaining GABA levels in the brain, achievable solely via the function of both SNAT-3 and SNAT-5

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Advanced glycation end products (AGEs) The content of AGEs was determined through spectrofluorometry

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Nakae et al

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Aggregation of vascular risk factors and risk of incident Alzheimer disease

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