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Consistent with this finding, we observed less respiration in response to CI and III substrates in isolated gWAT mitochondria and primary adipocytes from mice lacking adipocyte Ndufs2 (Extended Data Fig

J Dev Behav Pediatr 2010

This pro-ferroptotic lipid landscape facilitates membrane peroxidation, leading to cell death and the release of damage-associated molecular patterns (DAMPs)

Furthermore, the anti-apoptotic and cyto-protective effects of melatonin could stabilize mitochondrial membrane and help reset m -Dys 663
