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This higher prevalence is due to the increased rate of people carrying the pathogenic variant of the HADHA gene, responsible for the development of the disorder [8890]

J Pediatr Ophthalmol Strabismus 47:e1e3

This suggested that the CD36/MAPK/JNK pathway may play a key role in TMAO-induced foam cell formation ( in vitro study found that different concentrations of TMAO have no effect on the formation of foam cells in mouse macrophages ( The NLRP3 inflammasome is a polyprotein complex formed by the activation of PRRs that was recently reported to be crucial for the development of AS (Martinon et al., 2002

doi: 10.4331/wjbc.v10.i1.7