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Are mitochondria a permanent source of reactive oxygen species

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BSO treatment promoted an early induction of the senescence zone in wild-type nodules (Figures 8C,D) concomitant with an increase in the expression of the senescence-associated genes Cyp15a and TPP (Figure 7)

Laboratory testing of the gastric cancer tissue removed after gastrectomy demonstrated that allicin had the ability to impede cancer cell proliferation via apoptotic induction, cell cycle arrest (at G0/G1 phase) and modulated Bax and Fas (upregulated), Bcl-2 (downregulated) expression levels (Tilli et al., 2003)