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A., Shubin, V

(2015) reported that both TRPV1 knockout and pharmacological inhibition of the channel in an ex vivo model of increased IOP, resulted in fewer RGCs labeled for an apoptotic marker, suggesting that the entry pathway of TRPV1 Ca 2+ could be related to cell death (Sappington et al., 2015)

The key to both diagnosis and patient management lies in determining whether the pancytopenia results from a production disorder, a consumption disorder, or a combination of both [1]

However, dietary effects are context-dependent
