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Recruitment of BMDCs in glioblastoma can cause resistance to vatalanib treatment and the depletion of BMDCs can potentiate the effects of this anti-angiogenic drug [302]

Hypokalaemia (low blood potassium) in the first weeks of loading-dose treatment in patients who started severely deficient

In addition, the increased expression and activity of NOX and xanthine oxidase and NOS becoming uncoupled and structurally unstable also lead to increased ROS production [8]

Its clinical efficacy is largely limited by its pharmacokinetics due to low aqueous solubility, poor intestinal permeability and high first-pass catabolism in the intestine (Song I