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The peptide upregulates MMP-2 expression in dermal fibroblasts by 85%, facilitating breakdown of damaged collagen and enabling cellular migration through dense matrix environments, while simultaneously enhancing production of tissue inhibitors of metalloproteinases (TIMPs) to prevent excessive degradation

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These findings positioned FOXO4-DRI as a specialized research tool for investigating senescence-associated signaling, stress response pathways, and cell fate determination in aging models

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