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A KD switches primary energy consumption from glucose to ketones, bypassing the need to utilize the defective complex I and instead primarily utilizing complex II, which may help restore appropriate mitochondrial function and energy production (37)

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In highly specialised settings, a clinician might theoretically consider combining a GH-axis modulator with an incretin-based therapy for a patient with a specific, well-characterised metabolic phenotype for instance, significant visceral adiposity in the context of HIV-associated lipodystrophy who has not responded adequately to standard treatments

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