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This gene inhibits ferroptosis and is implicated in neurological disorders by modulating iron-regulated proteins such as transferrin, thus maintaining iron homeostasis within cellular iron pools (Ryan et al., 2023)

Listed mechanisms include IR serine phosphorylation states, IRS-1 modifications, PI3K/AKT pathway attenuation, GLUT4 expression changes, SNARE complex assembly defects, Rab GTPase cycling alterations, AS160/Tbc1D4 dysregulation, mTORC1/mTORC2 signaling perturbations, and inflammatory mediator effects (TNF-, SOCS3, IL-6)

The bioinformatics analysis using the GSE30979 dataset showed that compared with normoxic NSCLC patients, ALDH3A1 was upregulated in hypoxic NSCLC patients (Fig

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