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However, MNs cultured in the absence of B-27 demonstrated a significant increase in the mitochondrial H 2 O 2 level, which was, on average, four times higher than in the cytoplasm of the corresponding neurons

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The demonstration that the oxidative stress associated with mycotoxins can be related to direct suppression of GCLC gene function, post-translational modification of Nrf2 or the presence of excess TGF- suggests that the lack of glutathione may be due to the inability of mycotoxin-affected cells to adequately form glutathione