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The gsa2 strain itself is known to show a stronger GSAAT deficiency phenotype than its gsa1 counterpart (Sinha et al., 2022)
Additionally, the uremic milieu that results in elevated expression of TLR2 and TLR4 may be the reason why neutrophils and monocytes from CKD patients exhibit an excessive response to stimulation with lipopolysaccharides (LPS) (2, 87, 9095)
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Plakophilin-2 loss promotes TGF-1/p38 MAPK-dependent fibrotic gene expression in cardiomyocytes
