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2 The classic acute trigger is catecholamine signaling through beta-adrenergic receptors, which raises intracellular cyclic AMP (cAMP), activates protein kinase A (PKA), and drives PKA to phosphorylate HSL prompting its translocation to the lipid droplet where hydrolysis occurs

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Laboratories studying cartilage matrix biology, chondrocyte proliferation and differentiation, and growth factor signalling in musculoskeletal research models have incorporated this fragment as an experimental variable representing an expanding research frontier beyond its primary metabolic applications