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This precision medicine disarms the disease at the genetic level by hybridizing with SOD1 mRNA, orchestrating its degradation and consequently snuffing out the production of the detrimental SOD1 protein (8, 43, 44)

Furthermore, studies examining the sensitivity of various bacterial species to oxidative killing by neutrophils suggest that these organisms are not uniformly sensitive to the oxidative burst of these cells

363 Normal cells generate energy within the mitochondria, in the form of ATP, via the citric acid cycle and oxidative phosphorylation

However, many of these inhibitors, such as RSL3 and ML162, have exhibited suboptimal pharmacological properties in preclinical animal models, while others like BSO and auranofin have not yielded the desired anti-cancer effects in clinical trials, limiting their potential for clinical translation or application