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Quantitative proteomic analyses reveal that GPX4 downregulation during myocardial infarction contributes to ferroptosis in cardiomyocytes
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included 346,206 Korean subjects free of T2D who were followed over a mean of 9.2 1 years [40]
Neoplastic antigens can trigger a T-helper-mediated hypersensitivity reaction, activating monocytes and forming epithelioid histiocytes [28]
