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Notably, TMA-producing bacteria remained present after treatmentmeaning the mechanism was inhibitory (blocking the TMA lyase enzyme) rather than bactericidal, which avoids the collateral dysbiosis associated with antibiotic approaches

, Metabolism, 2010,

Given L-carnitine established role in facilitating mitochondrial -oxidation and its prior neuroprotective effects in adult ischemia models, it represents a biologically plausible candidate for investigation in neonatal brain injury

Hiraoka A, Arato T, Tominaga I: Reduction in blood free carnitine levels in association with changes in sodium valproate (VPA) disposition in epileptic patients treated with VPA and other anti-epileptic drugs