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The authors of these studies therefore concluded that the observed proinflammatory phenotype is likely mediated by increased TGF/NOX/ROS signaling within the HUVECs 23,30

[DOI] [PMC free article] [PubMed] [Google Scholar] 71.Bakunina N., Pariante C.M., Zunszain P.A

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doi:10.1016/s0040-4039(02)01762-8 Return to citation in text: [1] Yoda, H
