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(2003) found that hypothyroidism caused oxidative stress in the liver and heart, causing an imbalance between ROS and endogenous antioxidant body defenses, with subsequent damage to cellular macromolecules ( L-thyroxine administration moderated such oxidative stress in hypothyroid rats through normalizing the plasma MDA level and plasma TAC, in agreement with Varghese et al

second lieutenant on April 18, 1865

These processes can lead to large differences in the responses of isolated cells versus cells in intact organisms, emphasizing the fact that both basic and applied studies must ultimately rely on the use of whole animal models

Consequently, the metabolic capacity of the microbiota is disrupted, significantly impacting the bile acid (BA) pool (155)