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Specifically, this connection was hypothesized to occur through the gut dysbiosis-ocular surface-lacrimal gland axis which consists of five proposed immune-related mechanisms describing how ratio changes of gut commensal can lead to DES [114, 115]

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however, as the scar matures, the excessively compact ECM within the lesion core forms a rigid physical barrier that markedly hinders axonal regrowth (14, 15)

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