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Therefore, ROS accumulation increased by cisplatin induces Bax to cause mitochondrial dysfunction leading apoptosis, and ALA plays a central role in protecting mitochondria from this cisplatin-induced oxidative stress
This results in an increase in the level of intra-splenic Ach (119), which, in turn, leads to the inhibition of the release of TNF and other pro-inflammatory cytokines
The research experiment utilized a diabetic rat model fed with a high-fat diet
Rouault, T
