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Defining the roles for Vpr in HIV-1-associated neuropathogenesis
Expression of isoforms of NADPH oxidase components in rat pancreatic islets
Within the last decade, the brain was identified as a target organ for APAP toxicity in newborn laboratory mice based on profound, long-term loss of cognitive function observed following exposure to relatively low drug doses [12]
Glutaminolysis yields a metabolic by-product that stimulates autophagy
