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Recently, Naidu et al [80] demonstrated that ascorbyl stearate inhibited cell proliferation by interfering with cell cycle, reversed the phenotype and induced apoptosis by modulation of insulin-like growth factor 1-receptor expression in human brain tumor glioblastoma (T98G) cells

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KRAS mutations generally lead to the constitutive activation of downstream signaling cascades, such as MAPK and AKT pathways, which typically render CTX ineffective