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Description
Gut bacteria can convert the intake of choline into TMA, further generating TMAO in the liver, which reduces the bioavailability of choline and increases the lipid content of the newborn liver, leading to NAFLD and even nonalcoholic steatohepatitis
Mitochondrial deficits in human iPSC-derived neurons from patients with 22q11.2 deletion syndrome and schizophrenia
Ingestion of BB produces TMAO in a gut microbiotadependent manner in humans
A decrease in pup body weight was seen at 6 and 10 mg/kg which correlated with delays in developmental landmarks, such as preputial separation and vaginal opening
