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The possible mechanisms of arsenic-induced cardiotoxicity mainly include oxidative stress, DNA fragmentation, changes in cardiac ion channels (Figure 2, the possible arsenic exposure routes and mechanism of natural products against cadmium-induced cardiotoxicity have been summarized

Source: Koyuncu et al., 2025 Angiogenesis and Vascular Repair BPC-157 promotes VEGF (vascular endothelial growth factor) expression and the formation of new blood vessels

In vivo biodistribution indicated enhanced delivery of pioglitazone-loaded NLC into the brain after nasal administration, demonstrating the potential of NLC as a carrier for pioglitazone in AD treatment

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