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Most probably, given the relatively immediate effects BPC 157 therapy, its molecular pathways are not related to two major pro-inflammatory transcription factors activated in inflammation [91,92]: the activator protein-1 (AP-1) and nuclear factor kappa B (NF-B)

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The IL-7/IL-7R signaling axis increases the cell surface expression of Glut1 and glucose uptake by T cells (Fig

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