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however, the continuous accumulation of senescent cells, through the secretion of pro-inflammatory molecules known as the senescence-associated secretory phenotype (SASP), ultimately leads to chronic inflammation, tissue dysfunction, and age-related diseases.34 Senolytic therapies are designed to exploit the vulnerabilities of senescent cells, particularly their altered molecular mechanisms that heighten their susceptibility to particular stressors.37 Distinctive features include modifications in the expression of pro-survival proteins and activation of specific survival pathways

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Normalized expression (pg mg 1 ) = Cytokine concentration (pg mL 1 )/Total protein concentration (mg mL 1 )

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