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In conclusion, effective treatment of phosgene-induced lung injury involves early postexposure intervention that could reduce free radical species responsible for lipid peroxidation, correct the imbalance in the GSH redox state, and prevent the release of biological mediators such as leukotrienes, which are accountable for increased permeability

This hormonal cascade activates satellite cells, which are essential for muscle regeneration and the formation of new muscle fibers

Role of apoptosis repressor with caspase recruitment domain (ARC) in cancer

Activation of ATP citrate lyase by mTOR signal induces disturbed lipid metabolism in hepatitis B virus pre-S2 mutant tumorigenesis