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Interaction with insulin or oral diabetes medications may cause a significant drop in blood sugar levels

Lazertinib blocked the drug efflux function of ABCG2 and significantly increased the intracellular accumulation of doxorubicin, a known substrate of ABCG2 in the resistant S1-M1-80 cells in a dose-dependent manner

Furthermore, mitochondrial damage in myocardial cells of patients with DCM manifests as mitochondrial swelling, cristae disruption, and functional disturbances (32, 39)

From this large knowledge base, we only selected data that prove the regulation of signaling pathways in established BBB preclinical models both in vivo and in vitro (Table 4)