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In experimental models, blocking miR-34a restores SIRT1 activity, leading to increased TG degradation, increased -oxidation of fatty acids, and consequently a reduction in fat accumulation in the liver [302]
[DOI] [PubMed] [Google Scholar] 243.Spitz F., Gonzalez F., Duboule D
The results showed that GCLC was involved in the pathogenesis of Parkinson's disease and was associated with dopaminergic neuron deficits and motor deficits
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