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PPAR, -, and -, with PPAR being the primary cardiac transcriptional regulator of lipid homeostasis and the isoform most strongly implicated in DCM pathogenesis.26 PPAR exerts control over FA uptake, metabolism and storage through upregulation of genes coding for key proteins involved in these processes

Lipid peroxidation results in the formation of lipid peroxides and various toxic byproducts, which lead to a cascade of cellular damage, ultimately impairing sperm function

Mitochondria as a primary target for vascular hypoperfusion and oxidative stress in Alzheimer's disease

Five weeks of CLA supplementation (mixture of 39.4% c9, t11 and 38.5% t10, 12) has shown to increase lipid peroxidation, but had no effect on biomarkers of cardiovascular health, inflammation, or fasting blood glucose and insulin [20]