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For example, the NF-B p65 subunit can suppress NRF2s transcriptional activity by competing for coactivators, while NRF2-driven HO-1 expression can limit NF-B activation through carbon monoxide and biliverdin signaling [65,66]

Learn more about DMSA chelation therapy from the NIH PubChem resource

Three-dimensional-engineered bioprinted in vitro human neural stem cell self-assembling culture model constructs of Alzheimer's disease

1996;313 (Pt 1):1729
