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[2] This may stimulate the production of second messengers like cAMP and IP3 (inositol trisphosphate), which could propagate the signal within the cell and possibly activate protein kinases that may phosphorylate target proteins and regulate cellular processes

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ALC activated ERK signaling, increased histone acetylation, promoted PPAR nuclear translocation, and selectively enhanced mitochondrial respiration in mature OLs

194, 195 Whether this constitutes an off-target mechanism of resistance or rather reflects the inhibition of caspase-9 by AKT1-transduced signals 196 (hence representing a post-target effect) remains to be clarified