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In demyelinating environments, such as those induced by lysophosphatidylcholine injection, exercise significantly increases OPC proliferation and differentiation by activating the PGC-1 pathway, leading to thickened myelin sheaths and an increased number of remyelinated axons [301, 302]
Each region secretes its own set of hormones
However, these models are poor substitutes for in vivo models of atherosclerosis that involve different stimuli, immune populations, and timing
