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Description
Hearts from heterozygous (GPx1 +/ ) knockout mice were more susceptible to diastolic dysfunction following ischemia/reperfusion (I/R) injury than wildtype hearts [39] and GPx1 / hearts (from male mice) showed both contractile and diastolic dysfunction following I/R, with an increase in oxidative stress as measured by an increase in protein carbonylation [40]
HirschSPoniachickJAvendaoMCsendesABurdilesPSmokGet al
Fergusson MM
3 Circadian control of SAM-dependent NDUFS2 methylation, related to Fig
