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Integrative omics analyses reveal that MYC orchestrates ammonium metabolism reprogramming in prostate cancer through direct transcriptional control of GLS, with TCGA-PRAD data confirming co-amplification of MYC and GLS (q 20 mM) impair immune cell function by competitively inhibiting monocarboxylate transporters (MCTs), MCT1 in T cells and MCT4 in NK cells, leading to metabolic paralysis or apoptosis (180) ( Figure 4 )

Br J Clin Pharmacol 1984

Neurological damage: Promote neuroprotection and helps in recovery from neurological impairments

The reaction between DSF and hydrogen sulfide would generate HS-DDTC that will either decompose to DDTC or undergo further transformation