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Kanikarla-Marie, and J.A
Liu H, Zhao Z, Yan M et al (2023a) Calycosin decreases cerebral ischemia/reperfusion injury by suppressing ACSL4-dependent ferroptosis
In our South Florida and Doral patient population, where chronic UV exposure accelerates the cellular aging timeline faster than in most other US markets, the combination of Nrf2 antioxidant activation and epigenetic repair makes Epithalon a particularly well-matched tool for the local clinical context
Other relevant repair/oxidative stress protection mechanisms of NAC may also be relevant and worth exploring [33], including mechanisms by which NAC scavenges free radicals [34], induces neurogenesis [35], reduces mitochondrial apoptosis [36], reduces glutamatergic neurotransmission [29], and chelates metals as a form of oxidative stress protection [37]
