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Generally, MUFAs, such as oleic acid (OA), are synthesised from saturated fatty acids (SFAs) by the rate-limiting enzyme stearoyl-CoA desaturase 1 (SCD1), which is thus the primary determinant of MUFA levels

Like CaMKK2, the PKA signalling pathway is also considered to play a key role in the pathogenesis of bipolar disorder, as increased cyclic AMP levels and PKA activity have been frequently observed in post-mortem brains and peripheral cells of bipolar disorder patients [58,59,60,61]

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